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Association of LV dysfunction with MMP9 levels after MI with ST elevations

Basic information
Original title:Association of LV dysfunction with MMP9 levels after MI with ST elevations
Authors:Monika Pávková Goldbergová, Jiří Pařenica, Jolana Lipková, Nikolas Pávek, Petr Kala, Martin Poloczek, Anna Vašků, Ilona Parenicova, Jindřich Špinar
Further information
Citation:PÁVKOVÁ GOLDBERGOVÁ, Monika, Jiří PAŘENICA, Jolana LIPKOVÁ, Nikolas PÁVEK, Petr KALA, Martin POLOCZEK, Anna VAŠKŮ, Ilona PARENICOVA and Jindřich ŠPINAR. Association of LV dysfunction with MMP9 levels after MI with ST elevations (Association of LV dysfunction with MMP9 levels after MI with ST elevations). In Frontiers in Cardiovascular Biology 2012. 2012.Export BibTeX
@proceedings{986771,
author = {Pávková Goldbergová, Monika and Pařenica, Jiří and Lipková, Jolana and Pávek, Nikolas and Kala, Petr and Poloczek, Martin and Vašků, Anna and Parenicova, Ilona and Špinar, Jindřich},
booktitle = {Frontiers in Cardiovascular Biology 2012},
keywords = {MMP9 levels,polymorphisms, myocardial infarction with ST elevations},
title = {Association of LV dysfunction with MMP9 levels after MI with ST elevations},
year = {2012}
}
Type:Conference abstract
Keywords:MMP9 levels,polymorphisms, myocardial infarction with ST elevations

MMP9 from the family of matrix metaloproteinases (MMP) has a crucial role in processes of plaque formation, destabilization and rupture, and has been postulated as an important biomarker of acute coronary syndrome. Our results provide evidence that a higher level of circulating MMP9 is associated with worse EF, and AHF after STEMI treated by primary PCI and 30-days mortality. We found a significant association of TT genotype of MMP9 -1562C/T gene polymorphism with AHF, especially in male group. No significant influence of studied MMP9 gene polymorphisms on MMP9 levels was found.

 
 
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